As a licensed clinical psychologist, I've been in private practice in the Miami, FL area since 2010. My educational background includes a B.A. in psychology from Smith College, an MA and a Ph.D. in clinical psychology from American University, and training rotations at Children's National Medical Center, community clinics, and several different University Counseling Centers.
I specialize in helping children, adolescents, and young adults recover fully from eating disorders, depression, anxiety, and body image concerns utilizing evidence-based approaches including Family-Based Treatment (FBT) and Cognitive-Behavioral Therapy (CBT).
AFRID is a relatively new diagnostic category which was first added to the Diagnostic and Statistical Manual of Mental Disorders – Volume 5 (DSM-5) in 2013. ARFID is characterized by a pattern of avoidant or restrictive eating behaviors which led to significant nutritional, medical, developmental, and/or social-emotional consequences. Individuals with ARFID may restrict their food intake for a variety of reasons. Some individuals are hypersensitive to textures, tastes, and smells and feel comfortable with only a narrow variety of foods. Others don’t experience hunger cues, derive little pleasure or enjoyment from eating, and seem to have little interest in food. Still others begin restricting their food intake abruptly after a food-related trauma, such as choking, vomiting, or having an allergic reaction. Unlike those with Anorexia Nervosa or Bulimia Nervosa, patients with ARFID do not experience drive for thinness, fear of weight gain, or distorted body image.
The human body has evolved to protect itself from danger. The acute stress response, also known as the “fight or flight” response, describes the body’s physiological reaction to a perceived threat. In response to acute stress or danger, the body’s sympathetic nervous system is activated by the sudden release of hormones. In turn, the adrenal glands secrete hormones, including adrenaline and norepinephrine, which prepare the body for immediate action to fight off a predator (“fight”) or flee from danger (“flight”). The “fight or flight” response involves physiological symptoms intended to facilitate fighting or fleeing (e.g., rapid heartrate, increased blood pressure, increased oxygen flow to major muscles, often leading to shaking or muscle tension) as well as an alteration in psychological state associated with the perception of threat (anxiety, panic, aggression, violent impulses).
The third component of the acute stress response involves “freezing,” or defensive immobilization. This response may occur when a danger is perceived as life-threatening and inescapable. Think of a deer frozen in the headlights, or a cockroach on its back feigning death. A human victim of a physical attack or sexual assault may go still and limp rather than fighting back. The freeze response may, in some cases, allow the victim to survive, and in other cases, the numbness that accompanies it may spare the victim pain in his final moments of life.
Thousands of years ago, in our ancestral environment, we faced daily threats to our survival: saber-tooth tigers, wooly mammoths, cold, hunger, warring tribes. The acute stress response was essential to our survival. In today’s world, most triggers for anxiety are more subtle: public speaking, taking the SATs, career uncertainty, financial stressors. But our bodies don’t know the difference: the physiological and psychological responses are the same today as they were tens of thousands of years ago.
The fight, flight, and freeze responses are activated instantly and without conscious choice. Remember, these immediate reactions have evolved to protect us from danger in circumstances in which stopping to think could cost us our lives. We do not choose what our bodies perceive as dangerous or which branch of the acute stress response is activated.
Most people who suffer from anorexia nervosa have extreme fears of food, weight gain, or both. People with active anorexia nervosa who have not yet begun the recovery process are able to keep their fears within manageable limits through avoidance: avoiding “fear foods,” avoiding feelings of fullness, avoiding weight gain, avoiding the scale, avoiding the wrath of the anorexic voice by engaging in compensatory behaviors.
Recovery from anorexia nervosa requires sufferers to face these fears head-on multiple times every day. For these reasons, the bodies of people recovering from anorexia nervosa are locked in a near-constant acute stress response for months. This is why people recovering from anorexia nervosa usually feel much worse during treatment than they felt while they were acutely ill. The fight, flight, freeze response also explains most of the extreme and perplexing behaviors that we see in those who are recovering from anorexia nervosa.
The “fight” response is directed at the perceived source of threat. For a person with anorexia nervosa, the source of threat may be the person who serves him food (e.g., a parent), a treatment provider who requires weight gain (e.g., a therapist, physician, or dietitian), or the person’s own changing body. Therefore, it is common for people recovering from anorexia nervosa to physically or verbally attack their parents, to lash out at their treatment providers, and to turn their rage inward toward their own bodies as manifested by self-hatred, self-injury, or suicide attempts. Food itself is also a perceived source of threat. The fight response towards food may manifest physically as throwing food, cups, or plates; or it may manifest emotionally as a subjective feeling of hatred towards food.
The flight response in anorexia nervosa involves escaping the perceived source of threat. Many people with anorexia nervosa run away from the table at meal or snack times, lock themselves in their rooms, or run away from home for hours or days at a time to avoid eating. Some people jump out of moving cars, leave treatment appointments prematurely, or run away from treatment centers. In a more subtle manifestation of the flight response, many people with anorexia nervosa do their best to avoid the caregiver who is most firm about requiring full nutrition, and gravitate towards caregivers who are more lenient.
The “freeze” response entails some form of inaction or shutting down. For example, many people exhibit an inability or apparent refusal to speak about food, weight, or anorexia nervosa. Others have difficulty swallowing food. Some people freeze at mealtimes, unable or unwilling to pick up a fork or spoon for hours on end. In some cases, this acute food refusal necessitates spoon feeding or tube feeding. Some people with anorexia nervosa appear dissociated, “frozen,” or “zoned out” at mealtimes or at other times when the anorexic voice is particularly powerful. It is common for patients with anorexia nervosa to shut down during therapy appointments, avoiding eye contact and not engaging in conversation with their treatment providers.
These fight, flight, and freeze behaviors are extremely distressing for sufferers and for the loved ones who are supporting them. If you are supporting a family member through recovery from anorexia nervosa, please know that your loved one has not chosen to act or feel this way. These behaviors are extremely common, and temporary, reactions to the severe anxiety brought on by facing one’s worst fears multiple times per day. The fact that your loved one is exhibiting these acute stress responses is proof that his or her fears are being challenged. In fact, if the acute stress response is not being activated, there is a good chance that the anorexia nervosa is not being sufficiently challenged, which may result in a protracted course of illness and a delay in recovery. Remember to stay the course, to continue requiring full nutrition, prompt and complete weight restoration, and psychological support, regardless of the acute stress responses that come about.
Monday, September 28, is Yom Kippur, the Day of Atonement. Yom Kippur is considered the most important holiday in the Jewish faith and is typically observed by a day of fasting and religious services.
Every September, as Yom Kippur approaches, I engage in conversations with my Jewish patients and their families about observing this holiday. Families that are dealing with eating disorders frequently ask me if they, or their child, should fast for Yom Kippur. I believe strongly that anyone who has an eating disorder, is predisposed to developing an eating disorder, or has a history of an eating disorder, should not fast for any reason. So my answer, to anyone who falls into these categories, is a resounding NO!
Individuals with illnesses, including those with active eating disorders, are exempted, by Jewish law, from fasting. But what about those who have recovered from eating disorders, or those who are predisposed to eating disorders but have not (yet) developed a full-blown illness? Fasting poses a grave but hidden risk to these individuals as well.
Here’s why: most eating disorders are triggered by a negative energy balance, which is a period of time in which a person is consuming fewer calories than they are expending. People who are not predisposed to eating disorders may feel uncomfortable, weak, tired, or “hangry” while fasting, will overindulge in food to break the fast, and then will promptly return to their normal eating habits.
For people who have a genetic predisposition towards eating disorders, regardless of whether the eating disorder is currently active, a negative energy balance often takes a more sinister turn. These individuals often feel better when they are fasting, and fasting very quickly becomes a self-perpetuating cycle. The less they eat, the less they want to eat, until it gets to the point that they are not able to bring themselves to eat at all without significant external support.
In our culture, most eating disorders are initially triggered by an intentional negative energy balance such as dieting or a conscious attempt to “eat healthy” (which, sadly, is often a euphemism for dieting). But some eating disorders, especially in preadolescents and males, are initially triggered by an unintentional energy deficit through illness, dental procedures, athletic training, growth spurts, stress, depression, or religious fasting. Many people who recover from eating disorders will wisely avoid dieting, but experience relapses nonetheless through unintentional negative energy balances.
People who fast for religious reasons may have no intention of altering their body weight or shape, and may be truly devoted to observing their faith. But the body doesn’t know the difference. It simply registers that a negative energy balance is occurring and responds as it is genetically programmed to do. Once the switch is flipped by a period of negative energy balance, the eating disorder may be set in motion by a cascade of disordered thoughts and behaviors accompanied by increasing difficulty eating.
Some individuals in the Jewish community who have recovered from eating disorders eschew fasting on Yom Kippur and have found other meaningful ways to observe the holiday. For those who are predisposed to eating disorders but want to observe Yom Kippur, I often recommend a different type of fast. Perhaps a technology fast, such as going 24 hours without a smartphone or without social media. These types of fasts still convey a sense of deprivation, but without the dangerous possibility of triggering an eating disorder relapse. In fact, many people find that going without their phone or their social media for a day allows them to be more fully present in the moment and dramatically reduces their stress levels.
One of the most rewarding aspects of my job is the opportunity to engage with patients in long-term therapy. In my practice today, I have a number of patients who began treatment with me years ago, in adolescence, and are now in their 20’s. These patients first presented in my office with their parents during middle school or high school, suffering from severe eating disorders or depression or debilitating anxiety or, in some cases, all of the above. Some entered treatment kicking and screaming; others reluctant but resigned; still others wanting help and suffering desperately but requiring immense parental support to stay afloat.
In many cases, these adolescent patients received intensive Family-Based Treatment for six months or a year or more. In other cases, the teenage patients received individual Cognitive-Behavioral Therapy with active parental involvement. All of them made substantial progress in treatment. The majority recovered fully from their initial presenting diagnosis. Those who have not recovered fully are doing significantly better, living independent, fulfilling lives, but still experiencing symptoms and receiving ongoing care to keep their illness at bay. Now, years later, some of them continue with weekly therapy sessions. Others come in once or twice a month, or perhaps biannually like dental cleanings (we call this “mental hygiene”). Still others elect come in on an “as needed” basis, scheduling a few sessions here and there to help them cope with life events, navigate relationships, manage stress, or deal skillfully with bouts of depression or anxiety.
What unites these incredible young men and women is the fact
that they have taken full responsibility for their well-being. They have chosen to engage in long-term
individual therapy as an act of self-care.
Through their adolescent suffering, they have become acutely aware of
their susceptibility to mental illness.
They are choosing to receive therapy not only to prevent relapse of
illness but also to pursue optimal health.
Many of these young adults have
chosen to abstain from drugs and alcohol, even as their peers regularly smoke
pot and drink to excess. Many of them
have adopted consistent home practices of meditation or yoga. They make conscious, health-promoting choices
when it comes to sleep, nutrition, stress management, and physical activity. When faced with an important decision about
which graduate program to pursue, which job offer to take, which city to live
in, or even which person to date, they carefully consider the impact of these
choices on their quality of life.
Engaging in long-term therapy with patients like these
involves a number of gradual but significant transitions for all members of the
therapeutic relationship: the patient, the parents, and me. For
the parents and for me, there is the progression from the crisis management of
an acutely ill adolescent to the joy of stepping back into a supportive role
for young adult in his own quest for greater levels of well-being. The parents and I often begin our
relationship communicating multiple times per week to put out fires and to
ensure that we are in lock-step as we form a circle of safety around a suicidal
or eating disordered patient. As the
patient gradually assumes responsibility for her own well-being (which often
takes several years for those with adolescent-onset mental illness),
communication between parents and me subsides into an occasional email or phone
call. The patient is now a much
healthier, more mature young adult, and is trusted to schedule, attend, participate
meaningfully in her own therapy sessions.
In many cases, she pays for her own treatment as well.
The therapy itself goes through a significant evolution as I
shift from being directive and prescriptive, setting firm limits around
dangerous or debilitating symptoms, to engaging with the patient in deep
psychological work and collaborative goal setting. For the patient, there is the very welcome
shift from being told what she must do, in therapy and at home, to deciding
what issues are important to her and taking the initiative to seek support,
both therapeutic and familial, in achieving personally relevant goals. For the patient, this shift brings with it
a transformation from a defensive posture (as evidenced by panicking, shutting
down, or lashing out in therapy and at home) to a stance of openness and
receptivity (as evidenced by increased self-disclosure and self-awareness along
with the display of more vulnerable emotions).
I cannot begin to describe how rewarding it feels to support
a frightened, malnourished, deeply depressed teenager as she blossoms into a
healthy, confident, independent young woman who is attending college or
graduate school in another state, working at an exciting full-time job, getting
married, or giving birth to her first child.
It is fulfilling beyond words to join with young adult patients in the
journey of long-term wellness as they clarify their personal values, decide who
they want to be in this world, and take concrete steps towards achieving their
Those of you who have engaged in long-term therapy, either as a therapist, as a patient, know well how deeply personal and meaningful these relationships can be. There is a level of emotional intimacy that surpasses even that between spouses, between parent and child, or between the best of friends. In many ways, engaging in long-term therapy with self-motivated young adults is the polar opposite of Family-Based Treatment (FBT) for Adolescent Anorexia Nervosa. For most patients in my practice, the former would not have been possible without the latter.
Predisposing factors are important in helping individuals and their families understand their vulnerabilities and in alleviating the guilt, shame, blame, and stigma surrounding mental illness. Once families learn that the patient did not choose to develop this mental illness and the parents did not cause it, they have more energy to devote to recovery.
A word of caution: Predisposing factors are probabilistic, not deterministic. In other words, having one or more predisposing factors for a particular mental illness does not mean that developing that illness is inevitable. It simply means that vulnerability is heightened.
Precipitating Factors, known more casually as “triggers,” are events or circumstances that immediately precede the development of a disorder.
Examples of precipitating factors:
A loss of some sort (e.g., breakup of a romantic relationship, death of a loved one) often precipitates the onset of depression.
An energy imbalance (e.g., consuming fewer calories than one expends) almost always precipitates the development of anorexia nervosa or bulimia nervosa
A stressful situation (e.g., final exams in high school or college) may precipitate the onset of an anxiety disorder
Why are they important?
Awareness of the factors that have precipitated a mental health diagnosis is an important part of assessment. Relapse prevention planning, which typically happens towards the end of treatment, should help the patient and family develop awareness of the most common precipitating factors for their particular illness(es) so that they can avoid those precipitating factors when possible, or be prepared to approach them skillfully and mindfully, with ample supports in place, if they are unavoidable.
A word of cation: Precipitating factors are not the same as causes. The majority of individuals go through multiple stressors in their lives without developing a mental illness. A genetic predisposition is necessary, though not sufficient, for the development of a mental illness.
Another word of caution: Discussion of precipitating factors shouldn’t be a major focus of treatment. Once a disorder is set in motion by a perpetuating factor, the disorder takes on a life of its own and becomes self-perpetuating. It ceases to be “about” that precipitating factor.
Perpetuating Factors are events or circumstances that keep an illness in motion, or those things that cause symptoms to continue occurring over a period of time.
Examples of perpetuating factors:
Malnutrition, weight suppression, excessive exercise, and binge/purge behaviors perpetuate an eating disorder.
Environmental stressors, such as a highly rigorous academic environment, social exclusion or bullying, or elite athletic training, may perpetuate an anxiety disorder.
All forms of anxiety are perpetuated by heightened physiological arousal and avoidance.
Irregular sleep schedules, social isolation, and habitual use of marijuana are common perpetuating factors for depression.
Most successful mental health treatment is focused on identifying and modifying perpetuating factors. Most, though not all, perpetuating factors are modifiable and can be changed through cognitive or behavioral interventions.
A word of caution: The most powerful perpetuating factors are often those that directly impact physiology and brain function. For example, starvation is a powerful perpetuating factor in anorexia nervosa, and sleep deprivation is a powerful perpetuating factor in depression. A brain that is malnourished or severely sleep deprived is unlikely to respond well to psychological interventions. Cognitive perpetuating factors, which are also important, can be addressed most effectively later in treatment, after basic physiological function has been restored.
Prognostic Factors are factors which help to determine the eventual outcome, or prognosis, of treatment.
Examples of prognostic factors:
Early diagnosis and prompt intervention are positive prognostic factors.
Receiving evidence-based treatment is likely to shorten the duration of illness and increase the likelihood of achieving full recovery .
Dropping out of treatment prematurely reduces the likelihood of full recovery.
The presence of strong social support from family and friends increases the likelihood of full recovery and reduces the risk of relapse.
Full weight restoration, and maintenance of optimal body weight over time, dramatically improves the prognosis for anorexia nervosa.
Relapse prevention planning improves long-term prognosis by reducing the risk of relapse and guiding the type and timing of intervention if the patient begins to struggle again.
Practicing consistent self-care habits, including good sleep hygiene, regular exercise, and balanced nutrition, improves the prognosis for most illnesses.
Why are they important?
Prognostic factors are important to share with patients and families so that they can work together with treatment providers to create the best possible treatment outcome. Prognostic factors are relevant at the time of diagnosis (to help patients and their families act swiftly and choose evidence-based treatment), during treatment (to instill hope when treatment gets difficult and inspire everyone to stay the course rather than dropping out prematurely) and at the end of treatment, when relapse prevention plans are created. Prognostic factors are also important after treatment ends, as they relate to sustaining continued recovery and well-being.
A word of caution: prognostic factors, like predisposing factors, are probabilistic, not deterministic. Having positive prognostic factors does not guarantee a good outcome. Positive prognostic factors merely increase the statistical likelihood of long-term recovery and reduce vulnerability to relapse.
We have known for years that excessive smartphone use is correlated with depression. However, until recently, the direction of causality was not known. Does excessive smartphone use cause people to become depressed? Or do people who are already depressed use their smartphones more often, leading to unhealthy dependence? A recent study published in the Journal of Adolescent Health lends support to the former hypothesis. This study, which followed adolescents between the ages of 17-20, found that dependence on smartphones at the start of the study predicted depression and loneliness three months later.
Although the mechanism of action here is not yet known, I have several thoughts about why and how excessive smartphone use can lead to depression. First, there’s the neurobiological effect of the smartphone use itself, which activates the Sympathetic Nervous System (“fight or flight” response), increases threat perception, and disrupts sleep. The Sympathetic Nervous System (SNS) is designed to protect us from danger by elevating heart rate, increasing levels of cortisol, and giving us the burst of energy we need to fight off a predator or flee from danger. The SNS is meant to be activated periodically, and for limited durations of time, when danger is present until danger passes. It is not meant to be activated every few seconds, all day long, each time we receive a notification on our iPhone. Over time, chronic SNS activation leads to depletion and depression. So does sleep deprivation.
Second, there’s social comparison. Adolescents are especially vulnerable to peer influence and frequently compare themselves to others to gain a sense of their social standing. Teens who spend excessive amounts of time scrolling through social media are likely to judge themselves unfavorably compared to others. It is all too easy for these teens to conclude that other people are prettier, thinner, happier, more successful, and having more fun than they are. Of course, most people only post pictures of themselves looking great, having fun, and doing interesting things, even if the majority of their lives are spent looking average and completing mundane tasks like homework and chores. Nonetheless, upward social comparisons such as these tend to lower self-regard, and poor self-esteem fuels depression.
Finally, excessive smartphone use has to replace other activities. There are only 24 hours in a day. If a teen is spending 15 hours per day on his smartphone, what is he NOT doing? Well, for starters, he is probably not getting a good night’s sleep. He is probably not eating balanced meals with his family, or if he is, he’s eating mindlessly while scrolling through social media rather than engaging in meaningful conversation with his parents and siblings. He is probably not present or engaged in class, and he is constantly distracted while doing homework (if he even does homework), which means he is not learning much nor reaching his academic potential. He is not getting much physical activity or time outdoors. He may not be participating in social activities (of the in-person variety), or clubs, or hobbies, or lessons, or volunteer work, or religious services. In other words, the habits and activities that have been proven to help us stay healthy and balanced and socially connected, that give us a sense of meaning and purpose in life, are notably absent in those who are dependent on smartphones.
In my clinical practice, I strongly encourage teens and young adults to limit their screen time. To promote restful sleep, which is essential for both physical and mental wellbeing, I recommend turning off all electronic devices one hour before bedtime and leaving them off overnight. It is remarkable how much better teenagers sleep, and how much better they feel during the day, when they turn off their phones and computers by 10:00 pm, relax before bedtime, and sleep a full 8 hours.
For individuals who experience body image distress or social anxiety, limiting use of social media can help reduce symptoms. This could involve unfollowing certain people who are especially triggering, or simply limiting the amount of time spent on social media to 30 minutes per day, for example. I will never forget the 15-year-old girl who suffered from Generalized Anxiety Disorder who, upon getting rid of SnapChat for a week, experienced complete relief from her symptoms! Unfortunately, she later returned to SnapChat and began experiencing more anxiety. Eventually, she figured out a way to set limits on her SnapChat use, which allowed her to remain connected with her friends while causing only a slight increase in anxiety. Personally, I would have gotten rid of SnapChat completely and permanently, but try telling that to a very social, strong-willed 15-year-old.
Like many technological advances that preceded it (the automobile, the television, the internet), the smartphone is a wonderful invention that has improved the quality and efficiency of our lives. But there is a fallout from many technological advances, particularly when they are used carelessly or in excess (e.g., global climate change, couch potatoes, cyber-bullying). The smartphone is no exception.
WW International, the company formerly known as Weight Watchers, recently launched an app calledKurbo which is designed to help children ages 8-18 to diet and lose weight. The app is marketed as a “health coaching” tool, but a closer look at the company’s website reveals testimonials of children losing weight and dropping BMI points, complete with “before” and “after” photos. In recent years, the words “diet” and “dieting” have been replaced with words like “wellness” and “healthy eating” in popular nomenclature. But more often than not, when people refer to “eating healthy,” they are talking about restricting calories, reducing carbohydrates, and decreasing portion sizes. In other words, dieting in pursuit of weight loss. The brilliant marketing team at Weight Watchers, aware of this cultural shift in nomenclature, re-branded themselves as WW (Wellness that Works) to stay in vogue with their client base: people living in larger bodies.
But make no mistake: Kurbo is a diet app designed to help children lose weight. Although the app is touted as being based on years of scientific research, the very existence of this app defies the best available scientific evidence, which strongly suggests that CHILDREN AND ADOLESCENTS SHOULD NOT DIET.
Why? Let me count the reasons.
Bodies are meant to be diverse in size and shape. The very notion that a child or adolescent should lose weight in order to have an acceptable body flies in the face of genetics and natural size diversity.
Weight loss disrupts crucial physiological processes in the growing bodies of children and adolescents. Puberty requires significant weight gain to ensure proper development of the brain, bones, reproductive organs, and other vital body systems. Losing weight during adolescence can halt puberty, stunt vertical growth, and alter hormone levels.
Dieting is not effective at producing long-term weight loss, but it reliably predicts weight gain and depression. More often than not, dieting leads to weight cycling: losing weight in the short-term but regaining weight and ending up at the same weight, or a higher weight, in the longer-term. Our bodies have evolved to protect us against famine by slowing down metabolic processes when food is scarce (such as, when we are dieting and losing weight) and ramping up hunger signals and cravings, which often leads to overindulgence or binge eating. Weight cycling is associated with negative health outcomes, including increased risk of depression. Individuals who diet frequently experience cycles of shame, guilt, and feelings of failure each time they regain lost weight.
The normalization and glorification of diet culture is harmful and toxic to all children. When a person in a position of authority (e.g., doctor, parent, teacher, coach) tells a child or adolescent to lose weight, or places that child or adolescent on a diet, the message being sent (either subtly or overtly) is: “Your body is not acceptable as it is, and you must work very hard change your body in order to be attractive, healthy, happy, or socially accepted.” This message is damaging to a young person’s self-esteem, confidence, and body image.
Diet culture disproportionately targets and stigmatizes individuals in larger bodies, thus perpetuating weight stigma.
Although dieting itself does not cause eating disorders, dieting (or food restriction of any kind) can trigger the onset of an eating disorder in a child who is genetically vulnerable. Further, diet culture creates a toxic environment for individuals who are recovering from eating disorders. Eating disorders are dangerous, debilitating, difficult to treat illnesses that have the highest mortality rate of any psychiatric disorder.
Thankfully, there are many other individuals and organizations who share my sentiments on this matter and are publicly condemning this app.
Healthcare Providers Against Kurbo. A group of physicians, psychologists, therapists, and dietitians who specialize in treating eating disorders has formed a petition protesting the Kurbo app. You can read and sign the petition here.
So, you might ask, if dieting is not the answer, then what should we do about childhood obesity? The answer, based on the best available scientific research, is that obesity per se is not the problem, and thus the pursuit of weight loss per se is not the solution. Instead, adults who are charged with the task of caring for our youth (e.g., parents, teachers, coaches, and doctors) should encourage health-promoting behaviors in children across the weight spectrum. Children of all weights will benefit from eating balanced family meals containing a wide variety of foods from all food groups. Children of all weights benefit from adequate sleep, daily physical activity, and limited screen time. Children of all weights should be taught body acceptance and should be educated about size diversity. This is true health promotion. In some cases, these health-promoting behaviors will result in weight loss for higher-weight children, and in some cases, they will not. But regardless of what happens to the child’s weight, these health-promoting behaviors bring about genuine improvements in the child’s physical and mental well-being. And – this is important – NO HARM IS DONE.
I am honored to be spotlighted by LEAD, inc as a mental health advocate. LEAD is a nonprofit mental health advocacy organization founded in 2012 by a group of high school students. LEAD offers innovative, engaging, evidence-based programs which improve early intervention for mental illness.
As a clinical psychologist treating children, adolescents, and young adults, I am a strong proponent of early, aggressive, evidence-based interventions for young people who are struggling with mental illnesses. My personal and professional values align closely with those of LEAD, so I was thrilled when LEAD asked me to publish a VIP guest post for their blog! My post went live yesterday. This piece describes a topic about which I am passionate: Empowering Parents to Support Adolescent Eating Disorder Recovery.
Since opening my private practice in 2009, I have been privileged to work with over 300 individuals and families, providing consultation, evaluations, and treatment for a variety of mental health conditions. I believe in being transparent and straightforward about the services I provide and why I provide them. Individuals who are seeking mental health services for themselves or for their children have a right to know what treatment with a particular provider will actually be like, how long it will last, what outcomes they can expect, and what factors contribute to a more or less favorable outcome.
To this end, I collect detailed information on my patients’ treatment outcomes and publish the results on my blog. Here is an updated summary of treatment outcomes for the disorders I most commonly treat. For more detailed information on the types of treatment provided and treatment outcomes in my practice for each of these disorders, click on the category heading.
50% of patients who entered treatment with me completed a full course of treatment with me. 26% dropped out of treatment prematurely. 22% were referred to other providers who could better meet their needs. 3% moved to other geographic locations during treatment.
97% of patients who completed treatment achieved full remission. The remaining 3% achieved physical remission.
The majority of patients who completed treatment did so in a time frame of somewhere between 7 months and 2 years.
A full course of treatment required, on average, 27 sessions over the course of 17 months.
Patients with co-morbid conditions, such as anxiety disorders or depression, required more sessions, on average, than those without co-morbid conditions.
All patients who completed treatment achieved 100% full weight restoration, as indicated by a return to their pre-AN percentile patterns of growth for height and weight.
Average time to achieve weight restoration was 3.6 months.
Patients who recovered with individual therapy took longer, on average, to achieve weight restoration than those who recovered through Family-Based Treatment (FBT).
Patients receiving FBT were almost twice as likely to recover as those receiving individual therapy.
Patients receiving individual therapy were almost twice as likely as those receiving FBT to drop out of treatment prematurely.
Individuals with restrictive Anorexia Nervosa were twice as likely to achieve full remission as those with binge-purge Anorexia Nervosa.
For treatment drop-outs, there was a significant correlation between length of time spent in treatment and progress made. All treatment dropouts who were in treatment with me for at least 2 months had made significant progress towards treatment goals at the time of drop-out. Patients who dropped out of treatment after one month or less had not made any progress at the time of drop-out.
Over half of patients with bulimia nervosa (54%) discontinued treatment prematurely after making significant progress towards treatment goals, but prior to achieving full remission. 15% percent of patients were referred to other treatment providers or types of treatment that could better meet their needs, after making little or no progress in treatment with me. 8% of patients moved to other geographic locations and were thus referred to providers near their new homes. 23% of patients completed a full course of treatment with me.
Of those who completed a full course of treatment, 100% achieved full remission from their eating disorder.
A full course of treatment required, on average, 13 sessions over the course of 5 months.
Patients who took Prozac during treatment were more likely to achieve full remission than those who did not take medication.
Patients with a prior history of Anorexia Nervosa were less likely to recover from Bulimia than those who did not have a prior history of Anorexia Nervosa.
The presence of a comorbid diagnosis was not related to likelihood of achieving full remission from Bulimia.
Level of family involvement in treatment was not related to the likelihood of achieving full remission. This finding is in stark contrast to my outcomes for Anorexia Nervosa, in which family involvement was strongly correlated with positive treatment outcome.
Only 18% of patients who presented with a primary diagnosis of a mood disorder completed a full course of treatment with me. 50% discontinued treatment prematurely for unknown reasons, 15% moved to other geographic locations, and 18% were referred to other providers who could better meet their needs.
Of those who completed a full course of treatment with me, 83% achieved full remission from their mood disorder and the remaining 17% made significant progress towards their treatment goals.
Length of time to complete treatment varied dramatically (from 1 month to 3 years) based on individual needs, symptom severity, and progress. On average, a full course of treatment required 23 sessions over the course of 16 months.
High levels of family involvement predicted treatment completion and full recovery for adolescent patients but not for adult patients.
Patients who were self-referred were more likely to complete treatment and achieve full remission than those who were referred by another professional.
Predictors of less favorable treatment outcomes included hospitalization during treatment and taking psychotropic medication during treatment, most likely because these variables are markers for more severe forms of mental illness.
The following variables did NOT predict treatment outcome: age at intake, gender, ethnicity, duration of illness, diagnosis (type of mood disorder), presence of co-morbid diagnoses, rate paid for services, type of treatment received with me, or history of hospitalization prior to starting treatment.
Half of patients who entered treatment for anxiety disorders completed a full course of treatment with me.
Of those who completed a full course of treatment, 88% achieved full recovery and the remaining 12% made significant progress towards their treatment goals.
Length of time required to complete a full course of treatment varied dramatically from 1 month to 19 months, with a median treatment duration of 3 months.
Of those who discontinued treatment prematurely, 63% had made significant progress towards their treatment goals as of their final session with me, and the remaining 37% had made some progress.
Predictors of positive treatment outcome included high levels of family involvement in treatment, younger age at intake, shorter duration of illness, being self-referred to my practice, paying full rate for services, and having good attendance at therapy sessions.
Predictors of less favorable treatment outcome included presence of a comorbid diagnosis, taking psychotropic medication during treatment with me, and being referred to my practice by a psychiatrist.
Across diagnostic categories, less than half of patients who enter treatment with me complete a full course of treatment with me.
I tend to set the bar high for my patients, striving to engage them and help them continue to progress until they reach full remission.
Many individuals and families decide to discontinue treatment after making significant progress towards treatment goals, but prior to achieving full remission.
I hope that those who discontinue treatment after making significant progress, but prior to achieving full remission, continue to move forward and eventually achieve full remission with the support of their families and/or with other professional supports. My primary goal in working with families is to empower the parents to help their child. My goal is to become obsolete for that particular family. For this reason, when a family reaches a point where the parents are confident that “We’ve got it from here!” and my involvement is no longer necessary, then I have done my job well. Therefore, dropping out of treatment prematurely, after making significant progress in treatment, may not necessarily be a negative thing.
Across all diagnostic categories, patients who complete a full course of treatment with me do very well in recovery. The vast majority achieve full remission from their illness.
The following is an analysis of all patients who presented for treatment with a primary diagnosis of Bulimia Nervosa between the start of my private practice in 2009 and spring 2017. Given that this is an analysis of end of treatment outcomes, patients who were currently in treatment with me as of spring 2017 or later were not included in this sample. Patients and families whom I saw only for evaluations or consultations rather than treatment were not included in this sample.
Description of the Sample
This sample includes 13 individuals, all female, who were between the ages of 14 -27 at the time of their initial evaluation (mean age = 19). Duration of eating disorder prior to starting treatment with me ranged from 1 month to 11 years, with a mean duration of 3.5 years. Nearly half of the sample (46%) had a prior history of anorexia nervosa prior to beginning treatment for bulimia with me. More than ¾ of the sample (77%) had a secondary diagnosis, with the most common being Major Depressive Disorder, followed by ADHD and Anxiety Disorder Not Elsewhere Classified.
Description of the Treatment
Number of sessions attended ranged from 2 to 35, with a mean of 15 sessions. Duration of treatment ranged from 1 month to 28 months, with a mean duration of 8.8 months. Over half of the sample (54%) took psychotropic medication during their treatment.
All patients under the age of 18 had moderate or high levels of family involvement in their treatment. For patients over the age of 18, the degree of family involvement was determined by the patient’s living situation, family circumstances, and preferences. Among adults, 57% had no family involvement and the remaining 43% had some degree of family involvement, ranging from low (participation in at least one session and/or some phone contact with me outside of sessions) to high (participation in most or all sessions).
Approximately 62% of the sample paid full rate for services. The remaining 38% received a reduced rate based on financial need.
Status at End of Treatment with Me
Twenty-three percent of patients completed a full course of treatment with me. Of those who completed a full course of treatment, 100% achieved full remission. A full course of treatment resulting in full remission required an average of 13 sessions over the course of 5 months.
The rate of attrition (treatment dropout) in this sample was very high. Over half of patients in this sample (54%) discontinued treatment prematurely after making significant progress, but prior to achieving full remission. Fifteen percent of patients were referred to other treatment providers or types of treatment that could better meet their needs, after making little or no progress in treatment with me. Eight percent of patients moved to other geographic locations and were thus referred to providers near their new homes.
For complete definitions of terms such as “full remission” and “significant progress,” see this blog post from 2013.
Predictors of Positive Treatment Outcome
Completion of a full course of treatment was the strongest predictor of positive outcome. 100% of individuals who completed a full course of treatment achieved full remission.
Patients who took Prozac during treatment were more likely to achieve full remission than those who did not take medication.
Shorter duration of illness prior to beginning treatment with me was associated with greater likelihood of full remission. Patients who achieved full remission had been suffering from an eating disorder for an average of 3.3 years, whereas those who did not achieve full remission had been suffering for an average of 5.3 years.
A prior history of Anorexia Nervosa was associated with lower likelihood of achieving full remission from Bulimia Nervosa.